Disorders of Volition 在线电子书 pdf 下载 txt下载 epub 下载 mobi 下载 2024

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Sebanz, Natalie (EDT)/ Prinz, Wolfgang (EDT) 作者

Mit Pr

译者

2006-6 出版日期

504 页数

$ 56.50 价格

HRD

丛书系列

9780262195409 图书编码

Disorders of Volition 在线电子书图书标签: psychology

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Disorders of Volition 在线电子书 epub 下载 mobi 下载 pdf 下载 txt 下载 2024

Disorders of Volition 在线电子书 pdf 下载 txt下载 epub 下载 mobi 下载 2024

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Disorders of Volition 在线电子书著者简介

Natalie Sebanz is Assistant Professor in the Department of Psychology, Rutgers University. Wolfgang Prinz is Director of the Cognition and Action Research Unit at Max Planck Institute for Human Cognitive and Brain Sciences in Leipzig.

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Disorders of Volition 在线电子书 pdf 下载 txt下载 epub 下载 mobi 在线电子书下载

Disorders of Volition 在线电子书图书描述

Philosophers, psychologists, neuroscientists, and psychiatrists examine the will and its pathologies from theoretical and empirical perspectives, offering a conceptual overview and discussing schizophrenia, depression, prefrontal lobe damage, and substance abuse as disorders of volition. Science tries to understand human action from two perspectives, the cognitive and the volitional. The volitional approach, in contrast to the more dominant "outside-in" studies of cognition, looks at actions from the inside out, examining how actions are formed and informed by internal conditions. In "Disorders of Volition", scholars from a range of disciplines seek to advance our understanding of the processes supporting voluntary action by addressing conditions in which the will is impaired. Philosophers, psychologists, neuroscientists, and psychiatrists examine the will and its pathologies from both theoretical and empirical perspectives, offering a conceptual overview and discussing specific neurological and psychiatric conditions as disorders of volition. After presenting different conceptual frameworks that identify agency, decision making, and goal pursuit as central components of volition, the book examines how impairments in these and other aspects of volition manifest themselves in schizophrenia, depression, prefrontal lobe damage, and substance abuse.

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